Nutrigenomics, otherwise abbreviated as NGx, is the study of how genes interact with nutrients and dietary habits. It aims to understand how genetic variations affect individual responses to food, which can influence health, disease risk, and overall wellness. ¹
With our PRECISE Nutrigenomics assessment, you will gain personalized insights that empower you to make informed decisions about your diet and lifestyle. Our expert team analyzes your unique genetic blueprint to deliver personalized, actionable recommendations tailored to your body’s needs. From optimizing nutrition to enhancing overall well-being, this assessment puts you in control of your health journey — no guesswork, just precision. ²
Additionally, the resulting report from PRECISE NGx comprises three distinct sections:
This service evaluates 46 targeted gene variants to provide detailed insights into specific genetic alleles and their associated health implications. By understanding how these genes influence metabolism, nutrient absorption, and health risks, medical professionals can develop precise, personalized strategies for nutrition and supplementation. This tailored approach supports more effective interventions, helping individuals optimize their health and prevent potential issues through informed, evidence-based decisions. It’s a cutting-edge tool for aligning dietary and wellness plans with one’s unique genetic blueprint.
Certain food components like ethanol, caffeine, and lactose must go through biological processes to be utilized by the body. Genetic variations can influence how efficiently these components are metabolized, leading to differences in tolerance, absorption, and overall effects on health.
Caffeine is a natural stimulant that affects the brain. Its metabolism primarily occurs in the liver through the enzyme CYP1A2. However, some people carry a genetic variant that slows down CYP1A2 activity, classifying them as “slow caffeine metabolizers.” These individuals may be at higher risk of heart-related issues if they consume large amounts of caffeine daily.
Lactose, a carbohydrate found in dairy products, is broken down by the enzyme lactase, which is regulated by the LCT gene. People with certain genetic variations in LCT may produce less lactase, leading to lactose intolerance. This condition makes it difficult for them to digest lactose, often causing symptoms like bloating, diarrhea, and abdominal pain after consuming dairy products.
Certain genes play a role in how the body processes, utilizes, and stores nutrients. Variations in these genes may affect the body’s ability to convert, clear, or recycle specific nutrients, leading to differences in nutrient absorption and usage efficiency.
Alpha-linolenic acid (ALA), docosahexaenoic acid (DHA), and eicosapentaenoic acid (EPA) are essential Omega-3 fatty acids vital for heart, brain, and nervous system health. In the body, ALA can be converted into EPA and then into DHA, but this process is inefficient. Certain genetic variations in the enzymes responsible for these conversions may reduce the body’s ability to produce EPA and DHA from ALA, affecting overall Omega-3 levels. This can impact cardiovascular and cognitive health, making dietary intake of EPA and DHA from sources like fish more important for some individuals.
Beta-carotene is a red-orange pigment found in vegetables like carrots and pumpkins. In the body, it is converted into Vitamin A by the beta-carotene oxygenase 1 (BCO1) enzyme, which supports vision, immune function, and skin health. Certain genetic variants can slow this conversion, leading to an accumulation of beta-carotene in the body. As a result, individuals with these variants may experience skin discoloration (a yellow-orange tint) after consuming large amounts of beta-carotene-rich foods, a condition known as carotenemia.
The liver plays a key role in processing nutrients, food components, and medicines through conversion, storage, and detoxification with the help of enzymes. Certain genetic variations may affect the liver’s detox efficiency, potentially increasing health risks.
The Glutathione S-transferase Mu-1 (GSTM1) enzyme plays a key role in detoxifying harmful toxins, excess nutrients, and medications, aiding in their excretion from the body. This process helps protect against cellular damage and potential toxicity. Individuals with a genetic deletion of the GSTM1 gene have reduced enzyme activity, which impairs the body’s detoxification process. This genetic variation has been linked to an increased risk of certain cancers, such as lung cancer, due to the body’s reduced ability to neutralize carcinogenic substances and environmental toxins.
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